HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager Citing Articles Citing Articles via Google Scholar Google Scholar Articles by Fox, F. H. Articles by D.V.M., Search for Related Content PubMed PubMed Citation Articles by Fox, F. H. Articles by D.V.M.,

Clinical Diagnosis and Treatment of Ketosis

Ambulatory Clinic, Cornell University, Ithaca, New York 14850

ABSTRACT

Ketosis lactating dairy cattle of all ages, increasing during peak production is recognized and years. It may occur two to three weeks preparturn to four months Postpartum.common symptoms are diminished appetite, decreased milk, nervousness, profuse salivation, unnatural gait, licking themselves, grasping hard objects and damaging mouth, becoming explosively excited and unmanageable. clinically, animals reveal lack of alertness, eyes lack luster, rumen is hard and partially empty and noisy, feces are abnormally firm, and urine is clear, showing characteristics color responses to the Ross modification of the Rothera test. stress factors may cause ketosis such as high production indigestion, milk fever, partial starvation, metritis, mastitis, and ovarian cysts

Treatment for ketotic animals varies depending on conditions. Intravenous injection of dextrose is the author's standard treatment. Orally administered propylene glycol after initial use of dextrose or glucocorticoids has given excellent results. Intravenous or intramuscular cortisone has given extremely variable results. Intravenous or intramuscular adrenocorticotropin has given excellent results in prolonged ketosis, preceded by glucocorticoids. Dramatic recoveries have resulted from chloral hydrate for animals suffering from primary ketosis.